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First-episode psychosis is associated with measurable alterations in both central nervous system and peripheral biological systems.
Cholinergic projections connect key brain regions involved in cognition, memory, attention, and neurotransmitter regulation.
An integrated model links glutamate, GABA, and dopamine dysfunction to the development of cognitive deficits, negative symptoms, and psychosis.
Genetic vulnerability, environmental risk factors, and dopamine dysregulation interact to drive the development of psychosis.
Psychiatric disorders share substantial genetic risk, with particularly strong overlap observed between schizophrenia, bipolar disorder, depression, anxiety, and PTSD.
Childhood maltreatment can influence social functioning in psychotic disorders through multiple mediating and moderating factors.
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