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When a nerve impulse arrives at a noradrenaline nerve terminal, noradrenaline is released from synaptic vesicles into the synaptic cleft.
The mechanism of action of tricyclic antidepressants (TCAs) – a group of drugs used to treat affective, or ‘mood’, disorders.
When a nerve impulse arrives at a serotonin (5-HT) nerve terminal, 5-HT is released from synaptic vesicles into the synaptic cleft.
Depression is associated with reduced levels of the monoamines in the brain, such as noradrenaline. The selective noradrenaline re-uptake inhibitors are thought to restore the levels of noradrenaline in the synaptic cleft.
Depression is associated with reduced levels of the monoamines in the brain, such as serotonin (5-HT). The selective 5-HT re-uptake inhibitors (SSRIs) are thought to restore the levels of 5-HT in the synaptic cleft.
Depression is associated with reduced levels of the monoamines in the brain, such as 5-HT. The selective 5-HT and noradrenaline re-uptake inhibitors (SNRIs) are thought to restore the levels of 5-HT and noradrenaline in the synaptic cleft
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